TGFB1 and myelodysplastic syndrome: The results of knockdown studies reported that regulatory functions of either CD4+KO, CD25+KO or Foxp3+KO T regulatory cells were rescued by the presence of TGF-β1 secreting MSCs; a phenomenon present in myelodysplastic syndrome, where the elevation of TGF-β1, associated with cellular apoptosis, has similar effects [100].