TRAF3 and myeloid sarcoma: Moreover, the integration with protein–protein interaction pathways suggested new MS-susceptibility loci, including TNF-receptor-associated factor 3 (TRAF3), B cell membrane protein (CD48), B cell lymphoma 10 (BCL10), v-rel reticuloendotheliosis viral oncogene homolog (REL), and TEC protein tyrosine kinase (TEC) [32].