The positive correlation of the transcript levels among PRKD3, ERK1 and c‐MYC in the human breast cancer tissues was not significant, supporting that PRKD3 activated ERK1/c‐MYC axis independent of up‐regulating the transcript levels of the ERK1 and c‐MYC (Figure S4A). The gene discussed is MYC; the disease is breast carcinoma.