Interestingly, despite regulation of various transcription factors and pathways involved in inflammation including Stat1 in our mice, we did not find signs of kidney autoinflammation, i.e., vasculitis, tubulitis, or glomerulitis, in Sox2Cre::Pdgfrb+/K or Foxd1Cre::Pdgfrb+/J mice, even at advanced age. The gene discussed is STAT1; the disease is urogenital neoplasm.