Interestingly, despite regulation of various transcription factors and pathways involved in inflammation including Stat1 in our mice, we did not find signs of kidney autoinflammation, i.e., vasculitis, tubulitis, or glomerulitis, in Sox2Cre::Pdgfrb+/K or Foxd1Cre::Pdgfrb+/J mice, even at advanced age. Here, STAT1 is linked to medical procedure.