PDGFRB and atherosclerosis: Conversely, increased PDGFR‐β signaling resulting from activating mutations, gene translocation, or increased ligand abundance is involved in the pathogenesis of malignancies, atherosclerosis, and organ fibrosis (Bonner, 2004; Andrae et al, 2008; Demoulin & Essaghir, 2014; Klinkhammer et al, 2018).