Interestingly, despite regulation of various transcription factors and pathways involved in inflammation including Stat1 in our mice, we did not find signs of kidney autoinflammation, i.e., vasculitis, tubulitis, or glomerulitis, in Sox2Cre::Pdgfrb+/K or Foxd1Cre::Pdgfrb+/J mice, even at advanced age. This evidence concerns the gene PDGFRB and urogenital neoplasm.