FABP4 and neoplasm: Congruent with the IHC results showing the specific protein expressions in the tumors and lungs, and the in vitro co-culture experiments with stromal cells, this clustering analysis shows that CYP2C19, FABP4, and FABP5 expression and EET levels are influenced by stromal cells at the tumor and metastatic sites; furthermore, depletion of both lipid chaperones in TNBC cells are crucial to inhibit FABP-EET-mediated relapse or metastasis.