Given that some molecular functions, such as myoblast differentiation suppression via myoD expression changes or collagen type I synthesis regulation, are convergent for both excretory-secretory products of Trichinella ML and protein Mohawk, we postulate that Mkx can also play a role in muscle tissue remodeling being the host’s endogenous response to infection, however, the exact molecular pathway responsible for the Mkx expression pattern during trichinellosis is impossible to explain in these studies. Here, MYOD1 is linked to trichinosis.