UCK1 and acute myeloid leukemia: The CCK8 assay indicated that compared with the shcontrol cells, the MV4-11 cells with KLHL2 knockdown were more prone to inhibition, while the simultaneous loss of UCK1 abolished this inhibition (Figure 3A), suggesting that UCK1 loss mediates the effect of KLHL2 on 5'-AZA treatment in AML cells.