However, the same group verified that, contrary to the expected, ablation of GLUT1 attenuated apoptosis and increased drug resistance in triple-negative breast cancer cells (MDA-MB-231 cells), via upregulation of p-Akt/p-GSK-3β (Ser9)/β-catenin/surviving (Table 1) [52]. This evidence concerns the gene SLC2A1 and triple-negative breast carcinoma.