By examining the KEGG pathways, we noticed that the upregulated DEGs were enriched in DNA replication, the cell cycle, the nucleotide excision repair mechanism, the Fanconi anemia pathway, and DNA mismatch repair; the downregulated DEGs were mostly enriched in the ECM–receptor interaction, the PI3K–Akt signaling pathway, arginine and proline metabolism, Oligodontia-colorectal cancer syndrome, and Nevoid basal cell carcinoma syndrome (Supplementary Table 4). The gene discussed is AKT1; the disease is Fanconi anemia.