In addition, recent reports have shown a potential implication of pathogenic heterozygotic alterations of genes related to other DNA repair mechanisms such as non-homologous end joining (NHEJ), Fanconi anemia pathway (FANCM, FANCI, FANCL, FANCC, FANCB, FANCF, FANCE), base-excision repair (MUTYH), nucleotide excision repair (ERCC2, ERCC6), and mismatch repair (MLH1, MLH3, MSH2, MSH5, MSH6) (12, 14–16). Here, MUTYH is linked to Fanconi anemia.