Indeed, when this experimental setup was expanded to the A549 human alveolar epithelial cell line expressing FcRn but not FcγRI, FcγRIII, FcγRII, or FcαR there was an enhancement of CFU noted upon infection with Mtb opsonized with monoclonal IgG isotype and inhibition with the IgA isotype. The gene discussed is CD79A; the disease is infection.