Bakir-Gungor et al. reported new BD-associated common variants in pathways, including focal adhesion, complement and coagulation cascades, mitogen-activated protein kinases (MAPK) signaling, proteasome pathways, extracellular matrix (ECM)–receptor interaction, and transforming growth factor-β (TGFβ) signaling both in Japanese and Turkish populations (64). This evidence concerns the gene TGFB1 and Behcet disease.