Falzarano et al. (2016) additionally showed that truncated dystrophin is restored by in-framing with antisense oligonucleotides against exon 44 of DMD. Kim et al. (2016) demonstrated that myogenic reprogramming of urine cells derived from patients with DMD and limb-girdle muscular dystrophy (LGMD) type 2 could recapitulate the disease phenotype. This evidence concerns the gene DMD and Duchenne muscular dystrophy.