Pathway enrichment analysis indicated that the inversely downregulated genes by QSG were mainly enriched in myocardial fibrosis, apoptosis, and inflammation related pathways, such as Hippo, ECM-receptor interaction, focal adhesion, Wnt, PI3K-AKT, chemokine, JAK-STAT, NF-κB, and TNF signaling pathways (Figure 6(a)); the inversely upregulated genes by QSG were mainly enriched in energy metabolism related pathways, such as PPAR, fatty acid metabolism, and fatty acid elongation signaling pathways (Figure 6(b)). The gene discussed is SOAT1; the disease is Myocardial fibrosis.