TEXs can modify normal cells pheno- and genotype, not only by microRNA, but also oncogene EGFRvIII transfer or by enhancing mRNA expression for pro-angiogenic factors such as VEGF (vascular endothelial growth factor), HGF (hepatocyte growth factor), and IL-8, thereby enabling tumor cell adhesion to endothelium, resulting in metastasis [66–68]. The gene discussed is VEGFA; the disease is neoplasm.