Some tumor cells (including activated Myc) depend on glutaminolysis for survival and upregulate glutamine transporters and enzymes.9 Basal autophagy can be promoted by ammonia produced by glutaminolysis, protecting cancer cells from TNFα‐induced apoptosis and limiting proliferation under stressful conditions.10 Moreover, autophagy can interact with other pathways such as Ras and p53 in tumor cells to regulate tumorigenesis. The gene discussed is TP53; the disease is neoplasm.