ALB and metabolic dysfunction-associated steatohepatitis: In the absence of significant inflammation in the monoculture steatosis model, exposure to FFAs was previously reported to increase albumin production due to the over‐fed state of the hepatocytes.21 In the NASH model, with the presence of an enhanced inflammatory milieu, albumin levels were observed to slightly reduce over time, correlating with clinical observations, as albumin is a negative acute phase protein.28, 29, 30