Transcription of pro-inflammatory immune mediators (CxCL1/KC, CXCL2/MIP-2, CCL5/RANTES, TNF-α, IFN-γ and iNOS) and the fibrosis marker (ColA1) in the kidney were significantly more enriched in infected (NM, TD, IP) than uninfected mice (OM and controls). The gene discussed is TNF; the disease is ocular melanoma.