There was a significant link between AMH at the previously determined [45] cut off level (3.19 ng/ml) and FSHR variant rs2268361 in the control group (p = 0.016, Table 5).Multinomial logistic regression showed a significant association between the TT genotype of the rs2268361 variant and normal levels of AMH (<3.19) (OR = 6.2, B = 1.821, p = 0.009).Hence, homozygosity in the T allele ofrs2268361 is potentially protective, associated with normal levels of AMH among non-PCOS women. This evidence concerns the gene AMH and polycystic ovary syndrome.