The Th2 response in RSV is required for antibody production and to limit Th1 induced inflammation.5 Although studies in mice have demonstrated an important role for IL‐10 in limiting lung inflammation in RSV bronchiolitis,35, 36 the current evidence on the protective role of IL‐10 in RSV‐infected humans is conflicting.5, 37, 38 The production of IL‐10, IL‐4, and IL‐5 from in vitro stimulated T‐cells in our study was not statistically different between nonwheezers and wheezers, consistent with prior studies. Here, IL10 is linked to respiratory syncytial virus bronchiolitis.