A similar behavior was observed for SOX17, which was predicted as an oncogene in uterine corpus endometrial carcinoma associated with promoter peaks and as a tumor suppressor associated with hypermethylation in lung squamous cell carcinoma (Supplementary Data 7,, 8; Methods) These findings were confirmed by ChipSeq of SOX17 in endometrial cancer39, while SOX17 suppressed cell proliferation and promoter hypermethylation has been shown in lung cancer40. This evidence concerns the gene SOX17 and neoplasm.