LINC01018 and metabolic dysfunction-associated steatotic liver disease: Finally, to further determine the pathophysiological relevance of LINC01018 in human, we analyzed human liver RNA-seq data related to non-alcoholic fatty liver disease (NAFLD), and found that the expression levels of LINC01018 are significantly decreased in the livers of NAFLD patients compared to those in healthy control subjects (Supplementary Fig. 4a).