We initially found that miR-29a expression was increased but GAB1 was downregulated in lung tissues of neonatal mice following hyperoxia-induced BPD modeling, indicating that aberrant expression of miR-29a might be associated with the development of BPD miR-29a involves in both idiopathic pulmonary fibrosis cells and lung cancer cells from bronchoalveolar lavage (Bibaki et al. 2018). This evidence concerns the gene GAB1 and bronchopulmonary dysplasia.