LPL and hyperlipidemia: Liu et al. have shown in both mice and zebrafish that LPL activity is crucial for definitive haematopoiesis, liberating the polyunsaturated FA (PUFA) docosahexaenoic acid (DHA, 22:6) from TAG pools.43 Deleting apoc2, the gene encoding for an obligatory cofactor for LPL activity, attenuated the expression of Runx1, Cmyb, Beta‐globin and Rag1, indicating erythropoietic and lymphopoietic defects alongside with hyperlipidaemia.