One previous study [8] reported that PPARγ exerts anti-inflammatory effects through transrepression of the inflammatory activities of nuclear factor κB (NF-κB), signal transducers and activators of transcription 1 (STAT-1), and activator protein-1 (AP-1) via physical interaction, thereby influencing the occurrence and progression of sepsis. This evidence concerns the gene NFKB1 and Sepsis.