VEGFR2 was also localised in glandular and endothelial cells within all uterine cross sections; however, in slides from uterine tissues with stages 3–4 adenomyosis, the immunoreaction in glandular epithelium and vascular endothelium appeared to be stronger than for control slides and tissues from stages 1–2 adenomyosis (Fig. 5c, Table 1). The gene discussed is KDR; the disease is adenomyosis.