IL23R and candidiasis: Although similar levels of IL-23 were induced in response to S. aureus as with C. albicans, we could not recapitulate the phenotype of decreased myeloid cell survival that we observed in IL-23- and IL-23R-deficient mice during systemic candidiasis, indicating again that the role of IL-23 in preventing myeloid cell death was not conserved during systemic infections.