Moreover, treatment of AAA-ASCs with the autophagy activator, rapamycin, significantly reduced the levels of IL-6 and TNF-α and increased the level of IL-10 in the secretome, suggesting that autophagy may play a role in regulation of the immunomodulatory properties of AAA-ASCs (Figures 6(a)–6(c)). This evidence concerns the gene IL10 and triple-A syndrome.