A more detailed interrogation of their subcellular localization patterns is warranted using larger numbers of patients and controlling for the clinicopathological variables such as tumor size, tumor grade, and lymph node status, age of patient, etc. Nevertheless, the distinct localization patterns of p-Bad Ser112 and p-Bad Ser136 might be meaningful given that these phosphorylation events are also mediated by distinct kinases. This evidence concerns the gene BAD and neoplasm.