TGFB1 and Kaposi's sarcoma: These data also agree with the predictions from the analysis of a human KS-signature and newer RNA-sequencing studies derived from differentially expressed genes between KS and normal skin, which predicted the activation of several paracrine axes by upregulation of receptors and/or their ligands [2] and changes in TGFB signaling and glucose metabolism [43] similar to those observed in our MSC tumors.