A recent report has implicated SLAMF9 in the development of mouse pDCs, with a mild impairment in pDC maturation in the absence of SLAMF9 leading to the accumulation of immature pDCs in lymph nodes and a reduction in the percentage of interferon‐α‐producing pDCs after CpG ODN stimulation.30 Type I interferon signalling is known to contribute to macrophage necroptosis during Salmonella infection.35 Therefore, we might have expected enhanced clearance of Salmonella in Slamf9−/− mice. The gene discussed is SLAMF9; the disease is Salmonella Infections.