MYB and adrenal cortex carcinoma: We conclude that the Myb proteins with a 20 amino acid N-terminal deletion, as expressed in ACC tumors through the use of alternative promoter TSS2, have unique transcriptional activities, allowing ∆N Myb to activate and silence many genes that full-length Myb did not, including important genes involved in cell migration and perineural invasion that may correlate with worse overall survival.