In support of data also shown in Figure 4, the combination of TFV-1 with mAb AP2 did not induce FcγRIIa-dependent platelet aggregation or the time-dependent downstream ITAM/Syk/PLCγ2 pathway, which is associated with FcγRIIa-mediated platelet consumption and thrombocytopenia [11]. This evidence concerns the gene SYK and Thrombocytopenia.