Most recently, Scandiuzzi et al. (2010) have shown lower proteinuria, blood creatinine, blood urea nitrogen levels, and less severe renal histological damage in a model of glomerulonephritis in mMCP‐4 (chymase)‐deficient mice indicating an aggravating role of chymase in inflammatory kidney disease. Here, CMA1 is linked to glomerulonephritis.