Indeed, high levels of STING activators have been shown to lead the immune system toward sustained inflammation and consequent tumor initiation and progression.20 Furthermore, cGAS plays an important role in controlling cellular senescence a delicate cellular state vital for the elimination of pre‐cancerous state but also a reservoir of potentially harmful tumorigenic progenitors.31 Impaired STING expression may also allow the cancer cells to escape the immunosurveillance system. This evidence concerns the gene STING1 and neoplasm.