MALAT1 and dengue disease: In a DF model generated in immunodeficient NOD/SCID mice, transplantation of human MSCs transduced with null, or with antisense of miR-205-5p (as-miR-205-5p), or MALAT1 was compared, showing better therapeutic effects on DF recovery by MALAT1 overexpression than by miR-205-5p depletion, which seemed to be associated with improved vascularization on the disease site.