Moreover, it has been found that miR‐194 and miR‐200 by targeting BMI‐1 inhibit epithelial to mesenchymal transition (EMT) of endometrial cancer cells.7, 8 In contrast to results suggesting function of BMI‐1 in EMT and metastasis, Engelsen et al9 demonstrated that low, rather than high BMI‐1 expression is associated with an aggressive phenotype in endometrial carcinomas. Here, BMI1 is linked to endometrial cancer.