After infection, we observed significant enrichment of sortilin, IRAP, and GGA2 to the LCV, all known functional partners for intracellular GLUT4 sequestration (Li and Kandror, 2005; Shi et al., 2008; Shi and Kandror, 2005, 2007; Watson et al., 2004; Fig. 5, E–H). The gene discussed is GGA2; the disease is infection.