This phenomenon is mainly due to the participation of DKC1 in the production of telomerase and telomerase reverse transcriptase.11,12 The telomere length of patients with DC is significantly shorter than those of normal people.13,14 DKC1 also plays an important role in the processing of H/ACA small nucleolar ribonucleoprotein and is required for normal ribosome biosynthesis.15 The gene discussed is DKC1; the disease is dyskeratosis congenita.