Some mutations were shared among almost all multi-regional samples from a single patient (e.g., ZNF672 in Patient #28 (Fig. 3a); C1QTNF and MSS51 in Patient #29 (Fig. 3b)), suggesting that these genetic alterations were acquired early during adenomyosis development, whereas other mutations were more restricted (Fig. 3). Here, ZNF672 is linked to adenomyosis.