To sharpen our analysis, we examined the ClinVar database (https://www.ncbi.nlm.nih.gov/clinvar/), a public database of pathogenic gene variations, to determine whether any registered pathogenic/driver mutations of KRAS, PIK3CA, PPP2R1A, ARID1A, PTEN, and/or TP53 were detectable in our adenomyosis WES data, including in the small number of mutant reads that had been filtered out by our SNV criteria. The gene discussed is KRAS; the disease is adenomyosis.