To sharpen our analysis, we examined the ClinVar database (https://www.ncbi.nlm.nih.gov/clinvar/), a public database of pathogenic gene variations, to determine whether any registered pathogenic/driver mutations of KRAS, PIK3CA, PPP2R1A, ARID1A, PTEN, and/or TP53 were detectable in our adenomyosis WES data, including in the small number of mutant reads that had been filtered out by our SNV criteria. This evidence concerns the gene ARID1A and adenomyosis.