Confirming previous studies, we found that LINC00511 promoted GBM proliferation both in vitro and in vivo.26, 27, 28 Subsequently, we found that EMT‐related proteins, such as the mesenchymal markers Vimentin and N‐cadherin, were reduced, whereas the epithelial marker E‐cadherin was increased when LINC00511 was inhibited. This evidence concerns the gene CDH1 and glioblastoma.