Notably, we reported that Th2-mediated inflammation might contribute to the pathogenesis of OLP.[7] We also reported that CD4+ GATA3+ Th2 cells are sparse in affected lesions with CS.[4] Here, we showed direct evidence for the existence of CD4+ GATA3+ T cells in affected tissues from a patient with KD. The gene discussed is CD4; the disease is Cowden syndrome 1.