The oxidative imbalance responsible for liver tissue damage in diseases that affect the liver can be attenuated with sufficient ingestion of antioxidant micronutrients.24 One antioxidant that has been studied at length is vitamin E, which stabilizes mismatched electrons, prevents lipid peroxidation, reduces the tumor necrosis factor in alcoholic hepatitis, and prevents the activation of stellate cells, which are responsible for hepatic fibrogenesis.25 In the present study, all individuals presented with insufficient consumption of this nutrient. The gene discussed is TNF; the disease is alcoholic hepatitis.