SLC2A1 and hyperinsulinism: The control of SLC2A1 by ENTR1 in the context of the equine athlete is intriguing to speculate since SLC2A1 is expressed within equine lamellar tissue, and its expression is increased in hyperinsulinemia, therefore may play a role in the pathophysiology of equine laminitis (Campolo et al., 2016).