The most significantly altered pathways were hepatitis B, Epstein–Barr virus infection, human T-cell lymphotropic virus type 1 (HTLV-1) infection, Kaposi’s sarcoma-associated herpesvirus, nuclear factor-kappa B (NFKB) signaling pathway, hepatitis C, and tumor necrosis factor (TNF) signaling pathway. This evidence concerns the gene TNF and hepatitis A virus infection.