Following ischemia, levels of angiotensin 2 (AngII) increase (Ozacmak et al., 2007), which leads to vasoconstriction, increased oxidative stress (Kusaka et al., 2004), vascular endothelial failure and remodeling (Savoia and Schiffrin, 2007), the removal of NO, and apoptosis by AT1R. This evidence concerns the gene AGT and ischemia.