Overexpression of miR-31 rescued angiogenic and vasculogenic abilities of EPCs derived from patients with CAD by targeting thromboxane A2 receptor (TBXA2R) and FAT atypical cadherin 4 (FAT4), which in turn regulates VEGFA directly or indirectly via hypoxia-inducible factor (HIF)-1α [136]. The gene discussed is FAT4; the disease is coronary artery disorder.