Our results are also consistent with other studies, demonstrating that FOXE1 can interact with other factors and transactivate key genes in cancer such as SNAIL (Xu et al. 2015), an E-cadherin transcriptional repressor, or TERT (telomerase reverse transcriptase), which is coregulated by FOXE1 and the ETS factor ELK1 (Bullock et al. 2016). The gene discussed is SNAI1; the disease is cancer.