For example, USP9X stabilizes MCL‐1 and enhances tumour cell survival.33 USP7 deubiquitinates its substrate, the tumour suppressor CCDC6, and inhibiting USP7 activity determines CCDC6 degradation and sensitivity to PARP‐inhibitors treatment in tumour cells.34, 35 Here, we found that USP5 interacts with WT1 protein and stabilizes its expression. Here, MCL1 is linked to neoplasm.