Another striking example of this effect is mir-376a*, which targets different mRNAs under its edited and unedited form, i.e., autocrine motility factor receptor (AMFR) and Ras-related protein 2A (RAP2A) respectively, thereby promoting glioblastoma development and progression when a decreased editing capacity occurs in the brain [69]. The gene discussed is AMFR; the disease is glioblastoma.